Been getting quite into neuro stuff recently so thought id write this out . THIS IS COPIED OF MY OTHER ACC I AM NOT FRAUDING.
Depending on dosage, complete mitigation of side effects is not always possible. However, by using targeted interventions, neurological risk can be reduced as much as realistically achievable.
DHA
Glutathione
NAC
Astaxanthin
Carnosic acid
Vitamin C
High-dose melatonin may be implemented, ideally reserved for periods of very high androgen exposure.
Next, if concerns such as growth plate closure, fluid retention, or gynecomastia are secondary, estrogen should be maintained within range—preferably toward the higher end. Estrogen is critical for the brain: it supports synaptic plasticity, promotes neurogenesis, functions as a powerful neural antioxidant, and stabilizes neurotransmitter regulation
Androgens can be neurologically damaging, but with structured protocol design and informed compound selection, the magnitude of harm can be meaningfully constrained.
Depending on dosage, complete mitigation of side effects is not always possible. However, by using targeted interventions, neurological risk can be reduced as much as realistically achievable.
what actually happens to the brain under androgen exposure?
Androgens disrupt multiple neural signaling and maintenance pathways. The most relevant effects are outlined right here.
Decreased BDNF
Decreased neuroplasticity
Decreased neurotrophin-3
Decreased neuronal density
Decreased neurite outgrowth
Increased pro-inflammatory cytokines
Increased oxidative stress
These changes can significantly impair cognition by degrading learning capacity, attention, memory consolidation, problem-solving, executive function, and intrinsic neuroprotection. Risk is also elevated for depression, anxiety, ADHD-like symptoms, Alzheimer’s disease, Parkinson’s disease, and cerebrovascular events.
Androgens disrupt multiple neural signaling and maintenance pathways. The most relevant effects are outlined right here.
Decreased BDNF
Decreased neuroplasticity
Decreased neurotrophin-3
Decreased neuronal density
Decreased neurite outgrowth
Increased pro-inflammatory cytokines
Increased oxidative stress
These changes can significantly impair cognition by degrading learning capacity, attention, memory consolidation, problem-solving, executive function, and intrinsic neuroprotection. Risk is also elevated for depression, anxiety, ADHD-like symptoms, Alzheimer’s disease, Parkinson’s disease, and cerebrovascular events.
so what compounds can we use to reverse this damage?
- ACD85-6 binds to Trk-type receptors, increasing BDNF signaling, synaptic plasticity, neurogenesis, and neurotrophin-3, while simultaneously exerting anti-inflammatory effects.
- Pennylon upregulates the HSPA1 gene, improving insulin sensitivity and lipid handling not only systemically but within neural tissue as well. It strongly supports cellular resilience and enhances resistance to oxidative stress.
- SS-31 enhances mitochondrial efficiency, improving apoptotic regulation and enabling tighter control of reactive oxygen species.
DHA
Glutathione
NAC
Astaxanthin
Carnosic acid
Vitamin C
High-dose melatonin may be implemented, ideally reserved for periods of very high androgen exposure.
Next, if concerns such as growth plate closure, fluid retention, or gynecomastia are secondary, estrogen should be maintained within range—preferably toward the higher end. Estrogen is critical for the brain: it supports synaptic plasticity, promotes neurogenesis, functions as a powerful neural antioxidant, and stabilizes neurotransmitter regulation
Androgens can be neurologically damaging, but with structured protocol design and informed compound selection, the magnitude of harm can be meaningfully constrained.
